Tsu‐kung Lin - Neurology

Author Information

Tsu-Kung Lin is a prominent researcher in the field of neurology, currently affiliated with the Department of Neurology at Kaohsiung Chang Gung Memorial Hospital and Chang Gung University College of Medicine in Kaohsiung, Taiwan. His extensive work has significantly contributed to the understanding of various neurological diseases, particularly focusing on mechanisms involving oxidative stress, mitochondrial dysfunction, and neuroprotection.

Research Contributions

Dr. Tsu-Kung Lin has made substantial contributions to neurology research, with a particular focus on neurodegenerative diseases such as Alzheimer's and Parkinson's disease. His studies often explore the roles of oxidative stress and mitochondrial dysfunction. Notably, his work on the inhibition of 5-lipoxygenase has provided insights into potential therapeutic strategies for preventing retinal degeneration. Additionally, his research on mitochondrial quality control and ferroptosis underscores the complex interplay of cellular processes in neuroinflammation and neuroprotection. His investigations into antidiabetic agents for Parkinson's disease showcase his innovative approach to discovering cross-disease therapeutic strategies.

Aliases

Tsu-Kung Lin is also known by several variations of his name, including Tsu-kung Lin, Tsu–kung Lin, and Tsug-kung Lin. These aliases can be found across different publications and databases.

Publication and Citation Metrics

Metric	Value
Citation Count	4239
H-index	35
Paper Count	127

Publications:

5-Lipoxygenase Inhibition Protects Retinal Pigment Epithelium from Sodium Iodate-Induced Ferroptosis and Prevents Retinal Degeneration

DOI: 10.1155/2022/1792894

Year: 2022

Potential Roles of Sestrin2 in Alzheimer’s Disease: Antioxidation, Autophagy Promotion, and Beyond

DOI: 10.3390/biomedicines9101308

Year: 2021

Quality Matters? The Involvement of Mitochondrial Quality Control in Cardiovascular Disease

DOI: 10.3389/fcell.2021.636295

Year: 2021

Glucagon-Like Peptide-1 Receptor Agonist Ameliorates 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine (MPTP) Neurotoxicity Through Enhancing Mitophagy Flux and Reducing α-Synuclein and Oxidative Stress

DOI: 10.3389/fnmol.2021.697440

Year: 2021

Oxidative Stress, Mitochondrial Dysfunction, and Neuroprotection of Polyphenols with Respect to Resveratrol in Parkinson’s Disease

DOI: 10.3390/biomedicines9080918

Year: 2021

Oxidative Stress Biomarkers and Mitochondrial DNA Copy Number Associated with APOE4 Allele and Cholinesterase Inhibitor Therapy in Patients with Alzheimer’s Disease

DOI: 10.3390/antiox10121971

Year: 2021

Ferroptosis as a Major Factor and Therapeutic Target for Neuroinflammation in Parkinson’s Disease

DOI: 10.3390/biomedicines9111679

Year: 2021

Two Birds One Stone: The Neuroprotective Effect of Antidiabetic Agents on Parkinson Disease—Focus on Sodium-Glucose Cotransporter 2 (SGLT2) Inhibitors

DOI: 10.3390/antiox10121935

Year: 2021

Low Energy Shock Wave Therapy Attenuates Mitochondrial Dysfunction and Improves Bladder Function in HCl induced Cystitis in Rats.

DOI: 10.1016/j.bj.2021.06.006

Year: 2021

Mitochondrial haplogroups have a better correlation to insulin requirement than nuclear genetic variants for type 2 diabetes mellitus in Taiwanese individuals

DOI: 10.1111/jdi.13629

Year: 2021

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