Giampietro G. Schiavo - Neurology

Author Information

Giampietro G. Schiavo is a distinguished researcher affiliated with the Department of Neuromuscular Disorders at the UCL Institute of Neurology, University College London. His work is primarily focused on neurology, exploring various aspects of neuromuscular disorders, molecular mechanisms, and the development of therapeutic approaches.

Research Contributions

Giampietro G. Schiavo has made significant contributions to the understanding of neurological conditions, notably Amyotrophic Lateral Sclerosis (ALS) and Frontotemporal Dementia (FTD). His recent work includes pivotal studies on axonal mRNA transport, local translation, and the implications of TDP-43 mislocalization. Additionally, Schiavo has investigated the effects of potent human monoclonal antibodies for tetanus and developed imaging techniques for axonal transport. His research is critical for advancing the knowledge of molecular neuropathology and potential therapeutic interventions.

Aliases

Giampietro G. Schiavo is known by several aliases, including G Schiavo, G. Schiavo, and Giampietro Schiavo. These variations are often used interchangeably in scientific publications and databases.

Publication and Citation Metrics

Metric	Value
Total Citation Count	19,470
h-index	73
Total Paper Count	342

Publications:

Coupling axonal mRNA transport and local translation to organelle maintenance and function.

DOI: 10.1016/j.ceb.2022.01.008

Year: 2022

TDP-43 loss and ALS-risk SNPs drive mis-splicing and depletion of UNC13A

DOI: 10.1038/s41586-022-04436-3

Year: 2022

Kidins220 deficiency causes ventriculomegaly via SNX27-retromer-dependent AQP4 degradation

DOI: 10.1038/s41380-021-01127-9

Year: 2021

Common ALS/FTD risk variants in UNC13A exacerbate its cryptic splicing and loss upon TDP-43 mislocalization

DOI: 10.21203/RS.3.RS-386600/V1

Year: 2021

Exceptionally potent human monoclonal antibodies are effective for prophylaxis and therapy of tetanus in mice.

DOI: 10.1172/JCI151676

Year: 2021

Extremely potent Human Monoclonal Antibodies for the Prophylaxis and Therapy of Tetanus

DOI: 10.1101/2021.05.24.445390

Year: 2021

Rab10 regulates the sorting of internalised TrkB to retrograde axonal transport

DOI: 10.1101/2021.04.07.438771

Year: 2021

FUS-ALS mutants alter FMRP phase separation equilibrium and impair protein translation

DOI: 10.1126/sciadv.abf8660

Year: 2021

Expanding the Toolkit for In Vivo Imaging of Axonal Transport.

DOI: 10.3791/63471

Year: 2021

Molecular mechanisms of interneuronal transfer of pathogenic molecules

DOI: 10.1016/J.TOXICON.2020.11.458

Year: 2021

What Treatments are Available for Tourette Syndrome?

What Advances are Being Made in Neurocare Surgery?

How are neurological vision problems diagnosed?

What Are the Future Directions in AQP4 Research?

How is Cognitive Function Assessed?

What is Cognitive Impairment?

How do DMTs work?

What is the BDNF Gene?

How is Constipation Related to Neurological Disorders?

What is Progressive Relapsing Multiple Sclerosis (PRMS)?

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